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Low-Oxalate Diet

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Low-Oxalate Diet

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The basics

Low-oxalate diets are used primarily to control kidney stones. Approximately 1 in every 1,000 adults is hospitalized annually in the United States for kidney stones (renal calculi). Although kidney stones can be composed of different substances, more than 75% of the kidney stones in patients in the United States are made of calcium oxalate. All further mention of kidney stones in this section refers only to calcium oxalate stones. The information provided here does not apply to other forms of kidney stones.

  • Avoid high-oxalate foods such as beans, spinach, chocolate, wheat, and peanuts.
  • Drink plenty of fluids, limit your sodium intake, and consume adequate calcium (but do not take calcium supplements between meals).
  • Talk to a healthcare professional such as a doctor or a registered dietitian to make sure you’re following the best diet for your condition before you make any changes to your diet.

Best bets: Lean beef, pork, and lamb, vegetables like peas and lettuce, and cheese

Why do people follow this diet?

A hereditary condition can increase the risk of forming calcium oxalate stones. Intestinal resection, parathyroidism, and other more rare conditions also may cause renal calculi, and dietary factors can increase or reduce the risk of forming kidney stones. A low-oxalate diet is often prescribed for people who have increased levels of oxalic acid in their urine or who have a history of forming kidney stones. A treatment program including a low-oxalate diet and plenty of fluids reduces the risk of stone formation. A low oxalate diet is not always effective in reducing urinary oxalic acid levels because most oxalate found in urine is made in the body and does not come from the diet. People with a predisposition to stones are also encouraged to drink plenty of fluids (3 to 4 quarts [liters] per day), limit sodium intake, and consume adequate calcium, although calcium supplements should not be taken between meals.

What are the symptoms?

In combination with calcium, the oxalic acid crystallizes to form kidney stones—small pebbles that form in either the kidney or the bladder. If these stones are small enough, they will pass out of the body without being noticed. However if too large, they can cause severe pain, obstruction of the flow of urine, and sometimes infection in the urinary tract.

What do I need to avoid?

Scientists once thought that the greater the oxalate level in a food, the more likely it was to increase the risk of forming a kidney stone. However, researchers have discovered that consumption of only certain oxalate-containing foods is likely to significantly increase urinary oxalate. The foods reported by at least one group of researchers to cause a significant increase in urinary oxalate include spinach, rhubarb, beets, nuts, chocolate, wheat bran, strawberries, peanuts, almonds, and tea. Not every study has found tea to significantly increase urinary oxalate. There remains no universal consensus on which oxalate-containing foods belong on this list. Nonetheless, there is a growing awareness that the important issue for people with a history of kidney stone formation is to avoid certain high-oxalate foods—those that are most responsible for increasing urinary levels of oxalate.

To avoid oxalates ask about ingredients at restaurants and others’ homes, and read food labels. The following list is not complete. Consult with a healthcare professional before making any significant changes to your diet.

These foods are high in oxalate (greater than 10 mg per serving):

These foods are moderately high in oxalate (2 to 10 mg per serving):

Best bets

These foods are low in oxalate (0 to 2 mg per serving); eat as desired:

Are there any groups or books?

The following are some useful resources to help you learn more about dietary prevention and treatment of kidney stones.

The Kidney Stones Handbook: A Patient’s Guide to Hope, Cure and Prevention by Gail Savitz, Stephen W. Leslie, Gail Golomb. Roseville, CA: Four Geez Press, 2000.

Kidney Stones in Adults: National Kidney and Urologic Diseases Information Clearinghouse
www.niddk.nih.gov/health/kidney/pubs/stonadul/stonadul.htm

The Oxalosis and Hyperoxaluria Foundation
www.ohf.org

Bibliography

Alpers DH, Stenson WF, Bier, DM. Manual of Nutritional Therapeutics. 3rd ed. Boston, MA: Little, Brown and Company; 1995.

Berkow R, Fletcher AJ, et al, eds.The Merck Manual of Diagnosis and Therapy. 15th ed. Rahway, NJ: Merck Sharp & Dohme Research Laboratories;1987.

Brinkley, LJ, Gregory J, Pak Cy. A further study of oxalate availability in foods. J Urol 1990; 144:94–6.

Krieg C. The role of diet in the prevention of common kidney stones. Urol Nurs 2005;25:451–7 [review].

Mahan LK, Escott-Stump S. Krause’s Food Nutrition and Diet Therapy. 10th ed. Philadelphia, PA: W.B. Saunders Company; 2000.

Massey LK, Roman-Smith H, Sutton RA. Effect of dietary oxalate and calcium on urinary oxalate and risk of formation of calcium oxalate kidney stones. J Am Diet Assoc 1993; 93:901–6.


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